Porcine dynorphin A (1-13) is a potent, endogenous κ opioid receptor agonist and is antinociceptive at physiological concentrations.
In Vivo:
Dynorphin A (1-13) exposure (33 μM) causes a significant loss in neuronal viability at 4 h with a visible destruction in neuronal morphology seen at 16 h. Exposure to dynorphin A (1-13) causes acute increases in [Ca2+]i in individual neurons similar to increases seen with acute NMDA treatment. Continuous exposure to dynorphin A (1-13) (100 μM) causes a significant loss of neurons over time.
Related Prodcuts:
Deslorelin; Gonadorelin; Gonadorelin acetate; Sermorelin; Sermorelin Acetate; Etelcalcetide; DSIP; Lixisenatide; Angiotensin acetate; Epithalone; Buserelin; Cilengitide; Cilengitide trifluoroacetate; Tesamorelin; Selank; ACTH (1-39) Seractide; Ipamorelin